| dc.contributor.author | Kadarla, Vinay K. | |
| dc.contributor.author | Bigge, Brae Marie | |
| dc.contributor.author | Beck, Moriah R. | |
| dc.date.accessioned | 2017-09-18T02:08:23Z | |
| dc.date.available | 2017-09-18T02:08:23Z | |
| dc.date.issued | 2017-04 | |
| dc.identifier.citation | Vinay Kumar Kadarla, Brae Marie Bigge, and Moriah Rene Beck Myopalladin’s Role in Cardiac Muscle Function and Disease FASEB J April 2017 31:783.12 | en_US |
| dc.identifier.issn | 0892-6638 | |
| dc.identifier.other | WOS:000405461403695 | |
| dc.identifier.uri | http://www.fasebj.org/content/31/1_Supplement/783.12 | |
| dc.identifier.uri | http://hdl.handle.net/10057/14086 | |
| dc.description | Click on the URL link to access the article (may not be free). | en_US |
| dc.description.abstract | Decades of research have provided fundamental insight into the human heart’s structure and function. Yet, most cardiac malformations remain a mystery as scientists and clinicians continue to examine how inherited mutations and aging affect the normal biological functions of proteins associated with cardiac dysfunction. Recently, mutations in the muscle protein myopalladin have been linked to the pathogenesis of cardiomyopathy. | en_US |
| dc.description.sponsorship | Kansas INBRE under grant number P20 GM103418; The WSU Carl and Rozina Cassat Regional Institute on Aging Grant (2016); and Wichita State University. | en_US |
| dc.language.iso | en_US | en_US |
| dc.publisher | Federation of American Societies for Experimental Biology | en_US |
| dc.relation.ispartofseries | FASEB Journal;v.31:no.1 | |
| dc.title | Myopalladin's role in cardiac muscle function and disease | en_US |
| dc.type | Abstract | en_US |
| dc.rights.holder | Copyright © 2017 by the Federation of American Societies for Experimental Biology | en_US |
